Content area

Abstract

0.003 to 0.049 mmol per liter for benzoic acid and 0.60 to 1.060 mmol per liter for hippuric acid. Since discontinuing the use of bacteriostatic sodium chloride and bacteriostatic water in our nurseries in June 1981, we have seen no further cases of the gasping syndrome. Five additional premature low-birth-weight infants who were hospitalized in the neonatal intensive-care unit after we had stopped using bacteriostatic sodium chloride and bacteriostatic water and who were receiving mechanical ventilation and had umbilical arterial catheters in place were selected at random for blood and urine studies. Central-nervous-system symptoms were also described and ranged from sedation at low doses to convulsions and paralysis at higher doses.7 The neurologic symptoms seen in the present study -- including hypoactivity, hypotonia, coma, and seizure activity -- may have been due to a direct toxic effect of benzyl alcohol on the central nervous system. The clinical features of the gasping syndrome, the relatively large quantities of benzyl alcohol received by these infants, the positive identification of benzyl alcohol and its metabolites in their body fluids, and the known toxic effects of this bacteriostat suggest that benzyl alcohol was involved in the development of the gasping syndrome.

Details

Title
The Gasping Syndrome and Benzyl Alcohol Poisoning
Author
Gershanik, Juan, MD; Boecler, Betty, RN, MN; Ensley, Harry, PhD; McCloskey, Sharon, BS; George, William, PhD
Pages
1384-1388
Section
Medical Intelligence
Publication year
1982
Publication date
Nov 25, 1982
Publisher
Massachusetts Medical Society
ISSN
00284793
e-ISSN
15334406
Source type
Scholarly Journal
Language of publication
English
ProQuest document ID
1874438922
Copyright
Copyright Massachusetts Medical Society Nov 25, 1982